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Corrigenda

Volume 288, February 2005

Volume 57, February 2005

Pages F290–F297: Zager RA, Johnson ACM, Hanson SY, and Lund S. "Parenteral iron compounds sensitize mice to injury-initiated TNF- mRNA production and TNF- release" (http//:ajprenal.physiology.org/cgi/content/full/57/2/290; doi: 10.1152/ajprenal.00342.2004).

On p. F293, Figs. 3 and 4 were printed incorrectly. Correct versions are shown below.

View larger version (10K): [in this window] [in a new window]   Fig. 3. Plasma TNF- levels in mice subjected to glycerol-induced muscle injury with or without iv Fe treatment. Plasma samples were assayed for TNF- 3 h following glycerol injection alone or glycerol injection+FeS, FeG, or FeD. Plasma TNF- levels in normal mice are depicted. Glycerol alone caused a modest increase in TNF- levels (2.5 vs. 1 pg/ml for controls; P < 0.01). Concomitant Fe treatment dramatically increased the glycerol-induced TNF- increases, rising 2, 4, and 5x with FeD, FeG, and FeS treatments, respectively. [Note: there was far less TNF- generated in response to glycerol ± Fe treatment than was observed with LPS; see differences in y-axis values between Fig. 3 vs. the other figures].

View larger version (17K): [in this window] [in a new window]   Fig. 4. Comparison of iv vs. intramuscular (im) routes for FeG administration on LPS-induced TNF- generation. Left: mice received either iv or im FeG, or sham saline injections, and 24 h later their response to LPS administration was assessed. Prior iv Fe treatment caused a dramatic increase in LPS-mediated TNF- generation compared with non-Fe-pretreated controls. However, im Fe treatment did not affect LPS-generated TNF- levels. Right: same conditions as those depicted at left, except for a 48-h delay between Fe and LPS treatment. Again, iv, but not im, Fe sensitized mice to the LPS-mediated plasma TNF- increments.

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